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The redox/methylation hypothesis of autism: A molecular mechanism for heavy metal-induced neurotoxicity

Research output: Chapter in Book/Report/Conference proceeding › Chapter

Abstract

The rapid rise of autism rates over the past 25 years suggests a potential toxic effect from one or more environmentally encountered substances on the normal trajectory of neurological development, although increased diagnosis may also contribute. Two major autism categories can be identi ed, based upon whether or not individuals did or did not initially exhibit normal cognitive function and language acquisition.

Original languageEnglish
Title of host publicationAutism: Oxidative Stress, Inflammation, and Immune Abnormalities
Subtitle of host publicationOxidative Stress, Inflammation, and Immune Abnormalities
PublisherCRC Press
Pages113-130
Number of pages18
ISBN (Electronic)9781420068870
ISBN (Print)9781420068818
StatePublished - Jan 1 2009
Externally publishedYes

Publication series

NameAutism: Oxidative Stress, Inflammation, and Immune Abnormalities

Bibliographical note

Publisher Copyright:
© 2010 by Taylor and Francis Group, LLC.

ASJC Scopus Subject Areas

  • General Medicine

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